Search results for "Inclusion body myositis"

showing 7 items of 7 documents

205th ENMC International Workshop: Pathology diagnosis of idiopathic inflammatory myopathies Part II 28-30 March 2014, Naarden, The Netherlands.

2015

The idiopathic inflammatory myopathies (IM) are a heterogeneous group of diseases and diagnosis often necessitates a muscle biopsy. Five main entities are recognized: (1) dermatomyositis (DM); (2) polymyositis (PM); (3) necrotizing autoimmune myopathy (NAM); (4) sporadic inclusion body myositis (IBM); and (5) non-specific myositis. Other entities include granulomatous myopathy, macrophagic myofasciitis, and eosinophilic fasciitis (Shulman's syndrome). The pathological classification and subsequent identification of disease subgroups are extremely important for assessing treatment options and prognosis in the individual patient, yet classification criteria have not been standardized and vali…

2716 Genetics (clinical)medicine.medical_specialtyConsensusBiopsy10208 Institute of Neuropathology610 Medicine & healthPolymyositismedicineHumans2735 Pediatrics Perinatology and Child HealthColoring AgentsMyopathyGenetics (clinical)MyositisNetherlandsMuscle biopsyMyositismedicine.diagnostic_testbusiness.industryMusclesMacrophagic myofasciitisDermatomyositismedicine.diseaseDermatologyEosinophilic fasciitis2728 Neurology (clinical)Neurology2808 NeurologyPediatrics Perinatology and Child HealthPhysical therapy570 Life sciences; biologyNeurology (clinical)medicine.symptomInclusion body myositisbusinessNeuromuscular Disorders
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The cell-specific expression of metalloproteinase-disintegrins (ADAMs) in inflammatory myopathies

2007

Inflammatory cell invasion and cytokine activation are important steps in the pathogenesis of immune-mediated diseases of muscle. Metalloproteinase-disintegrins (ADAMs) are considered to play a critical role in leukocyte migration by promoting cellular adhesion, cleavage of molecules of the extracellular matrix and shedding of membrane bound cytokines. Here, we report the expression patterns of ADAM8, ADAM9, ADAM10, ADAM12, ADAM17 and ADAM19 in cultured human myoblasts and peripheral blood mononuclear cells (PBMCs) in vitro, as well as in biopsies from patients suffering from polymyositis (PM), dermatomyositis (DM), inclusion body myositis (IBM) and non-inflammatory controls. We observed an…

AdultMaleLeukocyte migrationBiopsyMyoblasts SkeletalMuscle Fibers SkeletalImmunologyT lymphocytesDown-RegulationGene ExpressionBiologyPeripheral blood mononuclear cellADAM19lcsh:RC321-571Downregulation and upregulationAutoimmune diseasemedicineHumansMyocytelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryCells CulturedAgedMyositisMacrophagesMiddle Agedmedicine.diseaseMolecular biologyADAM ProteinsMatrix metalloproteinasesNeurologyImmunologyFemaleInflammation MediatorsInclusion body myositisADAM9ADAM8Neurobiology of Disease
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Immune-mediated necrotizing myopathy is characterized by a specific Th1-M1 polarized immune profile.

2012

Immune-mediated necrotizing myopathy (IMNM) is considered one of the idiopathic inflammatory myopathies, comprising dermatomyositis, polymyositis, and inclusion body myositis. The heterogeneous group of necrotizing myopathies shows a varying amount of necrotic muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate. The underlying immune response in necrotizing myopathy has not yet been addressed in detail. Affected muscle tissue, obtained from 16 patients with IMNM, was analyzed compared with eight non-IMNM (nIMNM) tissues. Inflammatory cells were characterized by IHC, and immune mediators were assessed by quantitative real-time PCR. We demonstrate that immune- and non–immune-…

AdultMalePathologymedicine.medical_specialtyT cellBiopsyCell CountBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexReal-Time Polymerase Chain ReactionPolymyositisPathology and Forensic MedicineYoung AdultImmune systemSarcolemmamedicineHumansAgedAged 80 and overB-LymphocytesMyositisMacrophagesMusclesHistocompatibility Antigens Class IAutoantibodyImmunityComplement System ProteinsDermatomyositisMiddle AgedTh1 Cellsmedicine.diseaseCapillariesmedicine.anatomical_structureChild PreschoolImmunologybiology.proteinTumor necrosis factor alphaFemaleInclusion body myositisThe American journal of pathology
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Recent Advances in the Morphology of Myositis

1985

Summary Myositis in man may be divided into infectious and non-infectious forms. The myopathologist more often deals with the latter forms which comprise dermatomyositis/polymyositis, inclusion body myositis, mixed connective tissue disease/collagenoses, and granulomatous myopathies. Modern morphological techniques as enzyme-histochemistry, electron microscopy, immunohistology, and morphometry are of different value in various forms of myositis, but are often indispensable techniques in up-to-date diagnostic work up of a myositis.

Inclusion BodiesPathologymedicine.medical_specialtyGranulomaMyositisHistocytochemistrybusiness.industryImmunochemistryGranulomatous myositisCell BiologyDermatomyositismedicine.diseasePolymyositisDermatomyositisPathology and Forensic MedicineMixed connective tissue diseaseMuscular DiseasesVirus DiseasesmedicineHumansInclusion body myositisbusinessMyositisMixed Connective Tissue DiseasePathology - Research and Practice
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Value of insoluble PABPN1 accumulation in the diagnosis of oculopharyngeal muscular dystrophy.

2019

Background and purpose The aim was to assess the value of insoluble PABPN1 muscle fibre nuclei accumulation in the diagnosis of atypical cases of oculopharyngeal muscular dystrophy (OPMD). Methods Muscle biopsies from a selected cohort of 423 adult patients from several Italian neuromuscular centres were analysed by immunofluorescence: 30 muscle biopsies of genetically proven OPMD, 30 biopsies from patients not affected by neuromuscular disorders, 220 from genetically undiagnosed patients presenting ptosis or swallowing disturbances, progressive lower proximal weakness and/or isolated rimmed vacuoles at muscle biopsy and 143 muscle biopsies of patients affected by other neuromuscular diseas…

Pathologymedicine.medical_specialtyFluorescent Antibody TechniquePoly(A)-Binding Protein IOculopharyngeal muscular dystrophy03 medical and health sciences0302 clinical medicinePtosisMuscular Dystrophy OculopharyngealPABPN1 accumulations; PABPN1 immunofluorescence; oculopharyngeal musclular dystrophy; rimmed vacuoles; tubulofilamentous intranuclear inclusionsmedicineHumans030212 general & internal medicineMuscle fibreMuscle SkeletalCell NucleusMuscle biopsymedicine.diagnostic_testbusiness.industryExternal ophthalmoplegiaRimmed vacuolesmedicine.diseaseNeurologyCohortNeurology (clinical)Inclusion body myositismedicine.symptombusiness030217 neurology & neurosurgeryEuropean journal of neurologyReferences
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Myopathology of non-infectious inflammatory myopathies - the current status.

2007

Besides the classical inflammatory myopathies (IM), dermatomyositis (DM), polymyositis, and inclusion body myositis, the much larger spectrum of IM includes focal and nodular myositis, granulomatous myositis, macrophagic myofasciitis, graft vs. host myositis, eosinophilic myositis, and other immune-associated conditions, some of them only recently described. In addition, paraneoplastic, statin-induced and critical illness myopathies have been considered immune-associated IM. Infectious, i.e., bacterial, viral, and parasitic IM are much less frequent in the northern hemisphere. In IM, muscle biopsy is an essential diagnostic procedure to initiate therapy. The myopathological spectrum encompa…

Pathologymedicine.medical_specialtyMuscle biopsymedicine.diagnostic_testMyositisbusiness.industryMacrophagic myofasciitisCell BiologyDermatomyositismedicine.diseasePolymyositisImmunohistochemistryPathology and Forensic MedicineAtrophyEosinophilicImmunologymedicineHumansInclusion body myositisbusinessMyositisPathology, research and practice
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Glucocorticoid-sensitive hereditary inclusion body myositis.

1996

We report a hereditary muscle disorder with features of inclusion body myositis (IBM) in two adult sisters with slowly progressive asymmetrical muscle weakness. The findings of light microscopic and ultrastructural investigations of muscle biopsy specimens were consistent with a diagnosis of IBM. Both patients improved and stabilized on immunosuppressive treatment with corticosteroids and azathioprine. This differentiates our patients from other sporadic and familial cases of IBM. Clinical and histological features are described and compared with those of other previously reported families with IBM.

musculoskeletal diseasesPathologymedicine.medical_specialtyNeurologyeducationMuscle Fibers SkeletalAzathioprineMuscle disorderMyositis Inclusion Bodyparasitic diseasesmedicineHumansGlucocorticoidsMyositisImmunosuppression TherapyMuscle biopsymedicine.diagnostic_testbusiness.industryMuscle weaknessMiddle Agedmedicine.diseasePrognosisMicroscopy ElectronNeurologyFemaleNeurology (clinical)medicine.symptomInclusion body myositisbusinessGlucocorticoidmedicine.drugJournal of neurology
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